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Le composé innovant RCI001 pour la sécheresse oculaire attire à nouveau l'attention

2022-06-07

Health News: Innovative sécheresse oculaire candidat-médicament 'RCI1001' regains spotlight

Korean Medical Association News: Additional efficacy validation for innovant sécheresse oculaire candidat-médicament 'RCI1001'

Asia Today: Gachon University Gil Hospital draws renewed attention to innovant sécheresse oculaire candidat-médicament 'RCI1001'

Young Doctors: Professeur Kim Dong-hyun demonstrates efficacy of sécheresse oculaire candidat-médicament 'RCI001'

Medical Journal: Gil Hospital re-validates efficacy of innovant sécheresse oculaire candidat-médicament 'RCI1001'

Sports Chosun: Gil Hospital's Professeur Kim Dong-hyun provides additional proof of efficacy for innovant sécheresse oculaire candidat-médicament 'RCI1001'

'RCI001,' which inhibits Rac1, a cell signaling molecule, is a patented compound that improves the major symptoms of sécheresse oculaire notamment inflammation control, minimal eye irritation, and rapid recovery of épithélium cornéen.

Actuellement, steroid eye drops are used in divers ocular surface diseases notamment traitement de la sécheresse oculaires. Steroid eye drops have excellent effets anti-inflammatoires and provide superior short-term efficacy compared to other medications, but their long-term use is limited in actual clinical settings due to effets secondaires tels que increased pression intraoculaire and cataracts.

In this study, Professeur Kim's team confirmed the inhibitory effects of 'RCI001' on 'Rac1' and 'NLRP3 inflammasome (an inflammation-inducing immune protein).' First, topical treatment with 'RCI001' in an immune-overactivated LPS (Lipopolysaccharide) mouse model confirmed suppression of Rac1 expression and reduction of inflammatoire cytokine expression.

In an alkali-induced mouse eye injury model, 'RCI001' showed faster récupération des lésions cornéennes compared to conventional steroid formulations. De plus, it demonstrated reduced transcription and protein expression of Rac1 signaling molecules and the inflammatoire complex 'NLRP3' in the corneal tissue of this model.

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